...物、硫化物、磷化物等引起组织、细胞 中毒，氧利用障碍而引起的缺氧，称为组织中毒性 缺氧 缺氧性细胞损伤 (hypoxic cell damage) 主要表现为细胞膜、线粒体和溶酶体变化。
基于56个网页-相关网页
细胞形态呈现多样化，如圆形、梭形及椎形。 缺氧复氧损伤后心肌细胞伪足缩短或消失，折光性下降，搏动减弱或消失。
ResultsBeing injured by hypoxia and reoxygenation, refracting power of the cells declined, pseudo-podium became shortened or disappeared, cell beat became weak or stopped.
目的：观察新生大鼠缺氧缺血性脑损伤(HIBD)后海马区细胞增殖的情况，探讨脑组织内源性修复的可能机制。
Objective to observe the expression of proliferation in hippocampus following hypoxic-ischemic brain damage (HIBD) in neonatal rats and to explore the possible endogenous neurogenesis of brain.
结论急性低压缺氧可引起老龄大鼠大脑皮质和丘脑中et、NO含量明显升高以及大脑皮质血管内皮细胞和神经细胞明显损伤，ET、NO可能参与了缺氧性脑损害的病理过程。
Conclusion It is suggested that, the increase of ET and NO contents of cerebral cortex and thalamus may participate in the pathogenesis of hypoxia brain injury in old rats.
应用推荐