对我们而言，能够透过一次偶然的发现，将有关收缩、舒张和休克的细胞与分子机制等不同脉络的知识结合在一起，是件非常令人兴奋的事。
It has been very exciting for us to see the different threads of knowledge about the cellular and molecular mechanisms of constriction, dilation and shock come together through a chance observation.
探讨汉坦病毒感染对人胚肺细胞的影响，能为阐明汉坦病毒与宿主细胞相互作用的分子机制提供依据。
Study the effect of hantavirus on primary human embryonic pulmonary fibroblasts (HEPFs) will get evidence for the molecular mechanism of interaction between hantavirus and host cells.
这些研究表明疲劳产生机制的生理生化研究水平已进入亚细胞水平与分子水平。
These research results showed that Physiology and Biochemistry Studies on the making mechanism of sports fatigue have been on the sub-cellular and molecular levels.
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