这种生化阻止减少了老鼠的疼痛，这和采用药物治疗来达到提高组织中腺苷含量的效果是一样的。
This biochemical blockade reduced the animals' discomfort, as did treating them with drugs that boost the amount of adenosine in the tissue.
结果：实验药物组与模型组相比，小鼠全血、肝组织中的SOD活力增强，NO的含量减少，且呈剂量依赖关系。
Results: the experimental group compared with the aging model group, serum and liver tissue SOD activity increased, NO levels decrease, and show dose dependence.
结论：益智丹是治疗CHS的有效药物，其作用机制可能与改善记忆、提高脑组织DA含量有关。
Conclusion: YZD is an effective drug in treating CHS, its mechanism might be probably related to the improvement of the memory and the enhancement of DA receptor in cerebral tissue.
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