其起因是脂肪分解产物由于细菌酶的作用或皮脂腺分泌物的潴留而渗入周围组织，并从而引发了肉芽肿性炎症反应。
They develop when lipid breakdown products leak into the surrounding tissues from either bacterial enzymes or retained sebaceous secretions and incite a granulomatous inflammatory reaction.
然后细胞膜会变薄并破裂，释放蛋白质和酶，这两种物质能进一步分解周围组织。
This makes the cell membrane weak and it bursts, releasing proteins and enzymes which further break down the surrounding tissue.
他们的确有更高的乳酸脱氢酶起始水平。 这种酶与软组织分解相关，所以这个可能性不能被排除。
They did, indeed, have higher initial levels of lactate dehydrogenase, an enzyme associated with tissue breakdown, so this possibility should not be discounted.
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