目的：探讨缺氧大鼠脑线粒体能量代谢的特点。

AIM: to explore the characteristics of energy metabolism in brain mitochondria of rats exposed to acute and chronic hypoxia.

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这些结果说明线粒体能量代谢障碍是缺血再灌注损伤的始动环节，而氧自由基是造成损伤的主要因素。

These results showed that the energy metabolism dysfunction was the initial stage, and the damage of oxygen free radicals was the major factor in ischemia-reperfusion injury.

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目的：观察异丙酚对缺血再灌注损伤大鼠海马线粒体能量代谢、ATP酶活性、脂质过氧化及超微结构的影响。

AIM: to investigate the effects of propofol on the ATP content, ATPase activity, lipid peroxidation and ultrastructure in hippocampus mitochondrial in rats following global ischemia-reperfusion.

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