糖酵解的关键酶——丙酮酸激酶，如果它有遗传缺陷，就导致会atp缺乏、红细胞寿命缩短和溶血性贫血。
Inherited deficiency of pyruvate kinase, a key glycolytic enzyme, causes ATP deficiency, which leads to reduced RBC life span and hemolytic anemia.
糖酵解产生的丙酮酸在正常条件下被丙酮酸脱氢酶(PDH)转化成乙酰辅酶a (1 - 3)。
Pyruvate generated from glycolysis is converted to acetyl-CoA by pyruvate dehydrogenase (PDH) under normoxia (1-3).
来源于脂肪的甘油经两步酶促反应代谢转化为糖酵解的中间产物磷酸二羟丙酮。
Glycerol obtained from the breakdown of fat is metabolized by conversion to dihydroxyacetone phosphate, a glycolytic intermediate, in two enzyme-catalyzed reactions.
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