海马神经元损伤是神经突触可塑性调节中重要的一个环节，在抑郁症发病机制的研究中占有重要地位。
The damage of hippocampus neuron is the key part in plasticity regulation of synapse and play a critical role in the mechanism of depression.
目的观察大鼠脑梗死及电针干预后突触素(syp)的动态变化，探讨突触可塑性的物质基础、机制及针刺的影响。
Objective To explore the mechanism, material fundamental of synapse plasticity and acupuncture's efficacy by watching the alteration of SYP after cerebral infarction and acupuncture treatment.
已有广泛的研究表明突触可塑性如长时程增强和长时程抑制可以作为学习和记忆机制的细胞模型。
Extensive studies indicated that synaptic plasticity, such as long-term potentiation or long-term depression, is the cellular basis for learning and memory.
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