海马神经元损伤是神经突触可塑性调节中重要的一个环节，在抑郁症发病机制的研究中占有重要地位。
The damage of hippocampus neuron is the key part in plasticity regulation of synapse and play a critical role in the mechanism of depression.
研究人员指出，酒精并不是通过杀死神经元细胞导致失忆发生的。实际上，是这些产生的甾体物质影响了突触可塑性，造成了LTP和记忆形成方面的损害。
The scientists point out that alcohol isn't causing blackouts by killing neurons. Instead, the steroids interfere with synaptic plasticity to impair LTP and memory formation.
腹侧被盖区多巴胺神经元的突触可塑性的改变可能在许多药物滥用(包括可卡因滥用)所导致的回路修饰中起着一定作用。
Changes in synaptic plasticity in dopamine neurons of the ventral tegmental area (VTA) may contribute to circuit modification induced by many drugs of abuse, including cocaine.
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