目的探讨抗甲状腺药物(atd)引起粒细胞缺乏症的临床特点，治疗及转归。

Objective to investigate the clinical character, treatment and prognosis of agranulocytosis induced by anti-thyroid drug (ATD).

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抗甲状腺药物影响131i疗效的机理可能在于改变了131i的有效半衰期和(或)甲状腺对131i的摄取率。

Antithyroid drugs may influence the efficacy of 131i, the possible mechanism might be that antithyroid drugs change the effective half-time and (or) uptake of 131i.

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在接受抗甲状腺药物(atd)治疗的突眼性甲状腺肿615例患者中，215例(34.9%)发生毒性反应。

Out of 615 patients with Graves' disease who accepted the treatment of antithyroid drugs (ATD), 215 (34.9%) developed toxic effects of the drugs.

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