在炎症过程中，细胞被激活，同时数种炎症介质被释放包括。
During an inflammatory process cells become activated and several mediators including tissue factor (TF) are released.
PTCA后白细胞的激活可能触发一系列病理生理联级反应，释放某些炎症介质，促进血管平滑肌增殖导致再狭窄；
Activation of leukocytes after PTCA can trigger a serial cascade of pathophysiologic reaction, releasing some inflammation intermediates lead to restenosis.
炎症时释放的许多炎症介质都能够与TRPV 1发生相互作用，产生疼痛或痛觉过敏，并且通过各种不同的信号通路来调制TRPV1的活性。
In inflammatory pain, a number of inflammatory mediators are released, which modulate TRPV1 activity through various signal pathways or interact with TRPV1 generating pain or hyperalgesia.
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