通过激活休眠期的胚胎分子路径，干细胞被诱导参与新毛囊的形成。
By activating dormant embryonic molecular pathways stem cells were coaxed into forming new hair follicles.
主要讨论了UV - B信号途径及光受体，UV - B诱导的DNA损伤、转座子激活，UV - B对光合器官的分子伤害及相关基因表达的调控等。
UV B signaling pathway and specific UV B receptor, DNA damage, activation of transposons, molecular damage of photosynthetic apparatus and UV B regulation of related gene expression were included.
建立于目标基因激活和失活基础之上的几个化学分子诱导基因表达系统已有报道。
Various chemical inducible systems based on activation and inactivation of the target gene had been described.
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