这项工作显示了氧化低密度脂蛋白在骨桥蛋白基因表达中的作用,并且进一步解释了氧化应激在细胞因子调节中的作用。
This work demonstrates the role of OxLDL in the expression of the OPN gene and further highlights the role of oxidative stress in the regulation of this cytokine.
结论CHF患者较正常人运动耐受性降低,接近CHF患者日常生活活动的运动将短时间增强血浆可溶性细胞因子和氧化应激水平。
The six MWT, exercise mimicking the patients′ day to day activities, could increase plasma proinflamatory cytokine levels and oxidative stress in CHF patients for short time.
PH引起的氧化应激和炎症因子不仅阻碍肝细胞增殖,还可损伤肝微粒体CYP450酶。
Oxidative stress after PH can not only impede hepatocellular proliferation, but also damage liver microsomal CYP450.
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