n. crystallin
misc. lensprotein
这些数据表明晶状体蛋白的非酶糖基化可能会导致白内障。
These data suggest that nonenzymatic glycosylation of lens crystallins may contribute to cataract formation.
两者协同作用能有效地修复晶状体蛋白质和酶的巯基,维持晶状体的功能。
The two enzymes may work synergistically to repair thiols in lens proteins and enzymes and maintain the function of the lens effectively.
目的:研究半乳糖诱导白内障大鼠异常晶状体蛋白x 36的出现及其与白内障形成的关系。
Objective: to study the abnormal crystalline protein X36 in cataract SD rat and to explore the relationship be-tween X36 and cataractogenesis.
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