早老蛋白基因的突变是家族性阿尔茨海默病的主要病因。
Mutations in the presenilin genes are the main cause of familial Alzheimer's disease.
而且，我们发现突触前灭活早老蛋白而突触后未灭活时改变了突触的短期可塑性和突触易化。
Moreover, we found that presynaptic but not postsynaptic inactivation of presenilins alters short-term plasticity and synaptic facilitation.
任何人只要花上几百美元进行他们的基因组扫描，就可以决定是否看看他们的载脂蛋白e基因型有没有在增加他们患早老性痴呆的风险。
Anyone who has forked out a few hundred dollars to have their genome scanned can decide whether to find out if their APOE gene puts them at increased risk of Alzheimer's.
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