动物和人体实验均表明快速心房起搏或房颤可导致心房组织发生电重构,主要包括心房有效不应期(AERP)的缩短以及AERP正常的频率适应性消失。它与心房除极波的缩短、多发子波折返相关,有利于房颤的发生和维持。
基于190个网页-相关网页
...采用快速心房刺激致心房颤动动物模型,观察氯沙坦(血管紧张素受体I拮抗剂)对心房有效不应期（atrial effective refractory period,AERP）及缝隙连接蛋白40、45（Cx45、Cx40）的作用。
基于32个网页-相关网页
和心房有效不应期 atroal effective refractory period ; AERP
的右心房有效不应期 atrial effective refractory period ; AERP
的心房有效不应期 atrial effective refractory period
房颤时心房有效不应期 ERPAF
心房的有效不应期 atrial effective refractory period ; AERP
可使心房的有效不应期 atrialeffective refractory period ; AERP
结论腹主动脉结扎大鼠心房纤维化程度增加，P波时限增宽，IACT延长，心房有效不应期没有改变。
Conclusion The extent of atrial fibro sis was increased in abdominal aorta banded rats. Although longer P wave duration and IACT are present, there is no change in atrial effective refractory period.
结果显示：特发性房颤患者的心房有效不应期缩短，相对不应期延长，早搏刺激的房间传导延缓显著增加。
The results showed that patients with IAF had shorter atrial effective refractory periods and longer atrial relative refractory periods, and also longer interatrial conduction delays.
心房、房室结及心室相对不应期、功能不应期及有效不应期(erp)延长。
The relative refractory period, functional refractory period and effective refractory period (ERP) of right atrial, atrioventricular node and right ventricle were increased.
应用推荐