高血糖会导致大量的氧自由基产生，并破坏天然的抗氧化机制。
Hyperglycemia will cause overproduction of free radical and destroy natural antioxidant system.
在皮瓣缺血再灌注后产生大量的超氧阴离子自由基，其变化十分活跃。
A great number of superoxide anion free radicals were produced after ischemia and reperfusion of skin flap with active change.
心肌缺血再灌注损伤的主要机制为氧自由基大量释放，细胞内钙超载以及缺血导致的大量儿茶酚胺释放。
The mechanism of myocardium ischemia reperfusion injury involve in generating of reactive oxygen species, overload of Ca2 + and releasing of catecholamine by ischemia.
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