瘤苗放疗增敏作用机制是诱导细胞凋亡，增强NK和CTL活性。
The mechanism of vaccine radiosensitization includes induction of cell apoptosis and enhancement of NK and CTL activity.
目的探讨E1A基因对人宫颈癌细胞的放射增敏作用及其初步的作用机制。
Objective To investigate the radiosensitivity effect of E1A gene to human cervical carcinoma cell in vitro and its mechanism of action.
两者协同放射增敏作用的机制可能与凋亡有关。
And it indicated that the radiosensitizing effects of both drugs could be related to apoptosis.
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