...子刊-生物通 el(5q),是人类骨髓增生异常综合症(myelodysplastic syndrome, MDS)和急性髓细胞白血病(acute myeloid leukemia,AML)一种常见的克隆染色体异常,其上一个或多个肿瘤抑制基因的缺失被认为是引起这些疾病的致病机理。
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我们进行了一项临床试验旨在分析伏立康唑在急性髓细胞白血病化疗诱导阶段预防肺部浸润的有效性和安全性。
We conducted a trial to analyze the efficacy and safety of voriconazole in the prevention of lung infiltrates during induction chemotherapy for acute myelogenous leukaemia (AML).
在急性髓细胞性白血病aml和急性淋巴细胞性白血病all中,原始的急性白血病细胞进展、形成多达上亿个的白血病细胞。
In acute myelogenous leukemia (AML) and acute lymphocytic leukemia (ALL), the original acute leukemia cell goes on to form about a trillion more leukemia cells.
目的报道1例以CD7+和双重t(8;21)易位为特征的近四倍体克隆急性髓细胞性白血病(AML)。
Objective To report a case of acute myeloid leukemia (AML) with CD7 + near-tetraploid clones by doublet(8; 21).
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