结论：大鼠饥饿后早期确有肠黏膜组织结构受损，发生内毒素移位，同时伴有肠黏膜免疫学屏障受损。

CONCLUSION: the structure of intestinal mucosa is damaged in the earlier stage of starvation in rats, accompanied by endotoxin translocation and dysfunction of intestinal mucosal immune barrier.

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目的探讨重症急性胰腺炎(SAP)时血浆细胞因子与肠道屏障损害后肠源性细菌和内毒素移位的关系。

Objective to investigate the relationship between the plasma cytokines and the translocation of intestinal bacteria and endotoxin after gut barrier injury in severe acute pancreatitis (SAP) rats.

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