目的建立定量描述双侧椎动脉血流代偿性的方法。

Objective To establish the method to assess mathematically the compensation of flow in bilateral vertebralartery(BVA).

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结论：高血压时血管细胞为抵消或减轻et - 1等缩血管物质的过度分泌而代偿性的合成和分泌adm增多。

Conclusion: During the course of hypertension, vascular cell synthesize and secrete more ADM to compensate for the excessive secretion of et 1.

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像在呼吸性酸中毒时一样，也需要用许多复杂的公式来确定呼吸性碱中毒的预期代偿。

As in the case of respiratory acidosis, many complicated equations are used for determining the expected compensation in respiratory alkalosis.

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