当蛋白质结构层密度足够防止细胞渗透进人造血管时，研究人员会在其表面覆盖一层纤维原细胞片。
Then the researchers layered another sheet of fibroblasts on the outside of this scaffold, which is dense enough to prevent the cells from easily migrating to the inside of the engineered vessel.
之后将切片卷起促使细胞产生一种由结构支撑蛋白组成的混合物，即胶原及弹性蛋白。 纤维原细胞的问题在于它们会转化为平滑的肌肉细胞，最终将血管堵塞。
They then roll up the sheet and allow the cells to produce an interpenetrating mixture of structural support proteins, known as collagen and elastin.
结果VEGF是对内皮细胞特异的有丝分裂原，也有提高血管通透性的作用，在缺氧条件下作用尤为显著。
Results VEGF, as a mitogen specifically for endothelial cells, could strongly stimulate angiogenesis as well as vascular permeability, especially in hypoxia state.
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