大鼠糖皮质类固醇受体 Rat GR ; GR ELISA Kit
类固醇受体实验方法 Steroid Receptor Methods
类固醇受体超家族 steroid receptor superfamily
类固醇受体辅激活物 steroid receptor coactivator
小鼠糖皮质类固醇受体 Mouse GR ; Mouse glucocorticoid receptor,GR ; GR ELISA Kit
类固醇受体辅活化子 steroid receptor coactivators
类固醇受体激活物 steroid receptor activator
类固醇受体辅活化因子 Steroid receptor coactivator
类固醇激素受体的进化 Steroid Receptors Evolution of
目的观察小鼠烧伤后早期类固醇受体辅活化子(SRC)蛋白表达的时相变化,初步探讨烧伤早期糖皮质激素抵抗的分子机制。
Objective To observe the changes of expression of steroid receptor coactivators(SRC) during the early stage in the burned mice, and the mechanism of glucocorticoid resistance(GCR).
大多数雄激素非依赖性前列腺癌均高表达AR和核受体辅活化子、如转录中间因子2(TIF-2),ARA24、PIAS、类固醇受体辅活化子-1(SRC-1)等有力地支持了这个假说。
The "superactive AR"hypothesis was supported by the fact that the majority of AIPC expresses high level of AR and nuclear receptor(NR) coactivators, such as TIF-2, ARA24, PIAS, SRC-1.
结论RU486具有抗肿瘤作用,对不同类固醇激素受体状态的乳腺癌细胞其抗肿瘤作用的机制不同。
Conclusion RU486 has antitumor effect, but has different antitumor mechanism between steroid hormone receptor positive and negative breast cancer cell lines.
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