...13w末根据体重将高脂饲料组再分为饮食诱导肥胖(dietaryinducedobesity,DIO)和饮食诱导肥胖抵抗(dietaryinducedobesityresistant,DIO-R)组,比较各组大鼠能量摄入水平,血浆PYY3-36、NPY浓度,下丘脑匀浆NPY含量及下丘脑Y2-Rm..
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实验结束时，根据体重将高脂实验组大鼠分为饮食诱导肥胖(DIO)和饮食诱导肥胖抵抗(DIO R)大鼠，比较各组相关指标的差异。
At the end of the experiment, the high-fat diet group was divided into dietary induced obesity (DIO) and dietary induced obesity resistant (DIO-R) subgroups according to the final body weight.
同一种系大鼠中存在饮食诱导肥胖和胰岛素抵抗的个体差异，肥胖抵抗大鼠不易发生胰岛素抵抗。
There was difference of fat rich diet induced obesity and insulin resistance in the same species rats, the obesity prone rats had more tendency to become insulin resistance.
目的探讨高脂饮食诱导肥胖易感(OP)大鼠和肥胖抵抗(OR)大鼠，激素敏感性脂肪酶(HSL)和脂蛋白脂肪酶(LPL)基因表达的差别。
Objective To explore the gene expression of hormone sensitive lipase (HSL) and lipoprotein lipase (LPL) between obesity-prone (op) and obesity-resistant (or) rats induced by a high-fat diet.
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