结论：CCK-8能减轻LPS诱导兔离体胸主动脉反应性的异常改变，这可能是CCK-8抗内毒素休克作用的机制之一。
CONCLUSION: CCK-8 improved the vascular reactivities in the presence of LPS, which may be one of the anti-endotoxic shock mechanisms of CCK.
末次接种后两周检测各组T细胞对HSP60的反应和主动脉环的内皮依赖性舒张功能。
Two weeks after the last time inoculated, the response of T lymphocytes to HSP60 and endothelial-dependent diastolic function of aortic ring were detected.
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