Using the techniques they developed, researchers discovered that TLR4 proteins cluster in the membrane when confronted with LPS derived from e. coli, which increases cell signalling and response.
通过使用这种成像技术,研究人员发现TLR4蛋白质遇到来自大肠杆菌的脂多糖时会聚集在细胞膜内,提高细胞发出信号和反应的能力。
Rats were challenged by E. coli LPS to set up the model of endotoxic shock, AG group were treated by aminoguanidine.
用大肠杆菌内毒素(LPS)复制大鼠内毒素性休克模型,氨基胍治疗组采用氨基胍治疗。
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