Objective to study on the dexamethasone (dex) -induced mitochondrial depolarization and apoptosis process in mouse thymocyte.
目的研究地塞米松(DEX)诱导小鼠胸腺细胞凋亡进程中线粒体去极化作用特点。
Because there exist the depolarization effects in a majority of systems, the depolarization problem of system itself must be considered in the process of imaging.
由于大部分光学系统都存在消偏振效应，因此系统自身的消偏振问题在成像过程中必须考虑。
CONCLUSION: The process of thymocyte apoptosis mediated by NO in mice is successively PS ectropion, mitochondrial depolarization, CL oxidation and cell crenation.
结论NO介导的小鼠胸腺细胞的凋亡过程，依次为磷脂酰丝氨酸外翻、线粒体去极化、CL氧化及细胞皱缩。
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