Dopamine denervation in the striata of patients with Parkinson’s disease (PD) leads to changes in neural plasticity.
在帕金森病病人中,腹侧中脑多巴胺能神经元的死亡导致了纹状体的去神经支配,进而造成一系列病理变化,但是这些病理变化的分子机制并不清楚。
参考来源 - 脑发育和修复过程中功能基因的克隆和鉴定·2,447,543篇论文数据,部分数据来源于NoteExpress
The main mechanisms of treatment are atrial substrate modification and vagal denervation.
其主要治疗机制是改变房颤的心房基质和去迷走神经作用。
Damage to the neuromuscular junction following prolonged retraction can also lead to muscle denervation.
持续长时间牵拉造成神经肌肉接头处的损害亦导致肌肉去神经支配。
Denervation was achieved by multiple lesions at each level in an effort to provide effective denervation.
通过每个节段的多重损伤达到有效的去神经效果。
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