To examine this pathway in more detail, the researchers deleted the DDAH gene in mice.
为了更具体的验证这个途径，研究者把小鼠的DDAH基因敲除。
They also designed specific inhibitors (small molecules) which bind to the active site of human DDAH.
他们也设计了一个特殊的抑制物(小分子)，它可以结合在人类DDAH的活性位点上。
Genes and their pathways are crucial to our understanding of cardiovascular disease and a better understanding of DDAH-1 could lead to important new treatments.
探索这些基因和它们的途径，对我们了解心血管疾病和更好地了解DDAH - 1可导致新疗法的产生很重要。
应用推荐