They also designed specific inhibitors (small molecules) which bind to the active site of human DDAH.

他们也设计了一个特殊的抑制物(小分子)，它可以结合在人类DDAH的活性位点上。

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This study suggests for the first time that the loss of the activity of the enzyme DDAH-1 leads to reduced NO production and may cause heart and circulatory disease.

这个研究第一次说明ddah - 1酶活性的丧失导致NO产生的减少并且可能导致心脏和循环疾病。

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Scientists have hypothesised that if DDAH function is impaired, NO production is reduced, and that this could be an important feature of increased cardiovascular risk.

科学家假设，如果DDAH的功能被损害了，那么NO的产生也会减少，这可能是心血管疾病风险增加的一个重要特征。

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