In briefly, the development of myocardial remodeling in hypertension is a process that numerous active factors participated and co-regulated, and a result that collagenation exceeds its degradation.
简言之，高血压心肌重构的发生是一个由众多生物活性因子参与并共同调节的过程，是胶原生成大于降解的结果。
参考来源 - PPARα激动剂非诺贝特对自发性高血压大鼠心肌重构干预的实验研究·2,447,543篇论文数据，部分数据来源于NoteExpress
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