CONCLUSION Oxymatrine showed significant inhibition of voltage-dependent channel (VDC), which may be one of the mechanisms of counteracting arrhythmia.
结论氧化苦参碱对电压依赖性钙通道(VDC)具有明显抑制作用，这可能是其抗心律失常作用机制之一。
Chloride channel blockers, NPPB and ATP, inhibited the currents reversibly. Moreover, the inhibition of ATP on the outward currents was stronger than that on the inward currents.
氯通道阻断剂atp和NPPB可逆性地抑制此电流，ATP的抑制作用在外向电流显著强于内向电流。
The inhibition effect was irreversible at high dosage. Conclusion: blood resin inhibits the voltage gated sodium channel currents, which may well provide some analgesic effects.
结果：血竭剂量依赖地抑制单个DRG细胞电压门控性钠通道电流，高剂量的血竭抑制作用不可逆。
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