... cerebral ischemia attack ==> 脑缺血发作 cerebral ischemic reaction ==> 脑缺血反应 cerebral lesion ==> 脑损害 ...
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The pathophysiological mechanisms of ischemic cerebral injury involved in the release of excitatory amino acids, depolarization about the infarction, inflammatory reaction and neuron apoptosis.
缺血性脑损伤的病理生理机制包括兴奋性氨基酸的释放、梗死灶周围去极化、炎症反应及神经元凋亡等。
Conclusion Mild hypothermia has a protective role in cerebral ischemic tissue and inhibits the inflammatory cascade reaction, which may be one of the neuroprotective role mechanisms.
结论亚低温对缺血脑组织具有保护作用，对炎症级联反应的抑制可能是其发挥脑保护作用的重要机制之一。
CONCLUSION: GLUT1 expression is notably up-regulated in the penumbra region after focal cerebral ischemia, it may be a protective reaction against ischemic injury.
结论：GLUT1在缺血半影区的表达上调，可能是机体对缺血损伤的保护性反应。
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