CONCLUSION:The inflammatory cytokines can be released and the apoptosis of thymocytes are caused by negative-gram bacterial infection. The application of cefodizime and amikacin can significantly inhibit the thymocyte apoptosis and strengthen the immune systerm against infection.
结论：革兰阴性菌感染能诱导机体释放多种炎性细胞因子，并引起胸腺细胞发生明显凋亡，应用头孢地嗪、阿米卡星能显著抑制胸腺细胞的凋亡，增强宿主免疫系统抗感染的能力。
参考来源 - 肺炎克雷伯菌感染对小鼠细胞因子及胸腺调亡的影响·2,447,543篇论文数据，部分数据来源于NoteExpress
To evaluate the clinical effects of cefodizime on the patients with acute exacerbation of chronic obstructive pulmonary disease.
目的评价头孢地秦对慢性阻塞性肺疾病急性加重期的临床疗效。
Conclusion Cefodizime effectively improves the immune function of the host by regulating the balance between CD4 + cell and CD8 + cell.
结论头孢地嗪可通过调整CD 4 +和CD 8 +之间的平衡，有效地提高机体的免疫功能。
Objective To study the regulatory effect of cefodizime on the T-lymphocyte subsets of peripheral blood in mice with immunological liver injury.
目的观察头孢地嗪对免疫性肝损伤小鼠外周血T细胞亚群的调节作用。
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