细胞内钙稳态（calcium homeostasis）是一个复杂的生理过程，其依靠于质膜、内质网、线粒体等对Ca2 转运的调节。在正常情况下，细胞内总钙量的70%～80%存在于线粒体。
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The endoplasmic reticulum stress reaction resulted from calcium homeostasis unbalance and peroxidation damage after cerebral ischemia/reperfusion could down-regulate protein translation level and induce cellular apoptosis.
脑缺血再灌注后存在钙稳态失衡和过氧化损伤，由此可引起内质网应激反应，导致蛋白质翻译水平下降和诱发细胞凋亡。
参考来源 - 鼠脑缺血再灌后神经元凋亡的内质网应激机制及依达拉奉的影响The results showed that PI-PLC activities were decreased in the tumor cells after treated with verapamil. These changes occurred simultaneously with those changes indexes of calcium homeostasis.
结果表明经异搏定处理后的A_2细胞PI-PLC活性显著降低,并与钙恒稳的指标基本呈现相应的平行变化趋势。
参考来源 - 异搏定对小鼠肝癌腹水瘤细胞系PI·2,447,543篇论文数据，部分数据来源于NoteExpress
Vitamin d, calcium homeostasis, and skeleton accretion in children.
维生素d，钙稳态，儿童骨架增大。
It is suggested that cadmium might cause apoptosis and disequilibrium of calcium homeostasis in SD rat kidney cells.
镉能引起离体sd大鼠肾细胞的凋亡并引起胞内钙稳态的失调。
Objective: to study heart mitochondrial calcium homeostasis during ischemia - reperfusion under the protection of cardioplegic solution.
目的：观察心肌在高钾停搏液保护下经历缺血再灌注损伤时，其线粒体钙稳态的变化。
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