A high intensity of COX-2 also predicted biochemical failure, the immediate PSA rise after treatment.
COX - 2的高强度也预示着治疗后的生化紊乱与PS A水平升高。
The final step was to demonstrate that the proteins associated with those genes have an actual role in organ failure via real-world biochemical tests.
最后一步是通过有效的生化试验证实与这些基因相关的蛋白在器官衰竭中确实起着作用。
ConclusionThe plasma BNP concentration appears to be a biochemical marker of an increased risk of all-cause mortality in patients with heart failure, especially multiple organ dysfunction.
结论BNP水平是心力衰竭患者死亡危险增加的一个生化指标,是影响患者预后的一个重要危险因素。
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