目的：从黏膜免疫及炎症相关细胞因子的角度探讨脾虚痹证的发病机理。

Aims: to explore the pathogenesis of Bi-syndrome (arthritis) superimposed on spleen deficiency by investigating the changes of mucosal immune functions and inflammation-related cytokines.

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用免疫组化方法检测胃黏膜细胞的凋亡和增殖以及凋亡相关基因BCL-2、BAX的表达。

We used immunohistochemistry to detect the gastric mucosal cells apoptosis, proliferation and the expression of BCL-2, BAX.

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