在应答缺血性心肌损伤时，脾单核细胞能动性增加，全体离开脾，在受伤的组织聚集，参与伤口愈合。
In response to ischemic myocardial injury, splenic monocytes increase their motility, exit the spleen en masse, accumulate in injured tissue, and participate in wound healing.
观察结果发现脾脏具有储存单核细胞和快速部署的作用，而且确认了脾单核细胞是一种可用于调节炎症的资源。
These observations uncover a role for the spleen as a site for storage and rapid deployment of monocytes and identify splenic monocytes as a resource that the body exploits to regulate inflammation.
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