结论CSS和CH单独作用均为肿瘤启动剂；当二者联合作用时可协同启动肿瘤的发生。
Conclusion Both CSS and CH may act as initiator in carcinogenesis, and may synergistically initiate tumors when CSS and CH were exposed simultaneously.
因此应用药物促使肿瘤启动细胞凋亡程序，就成为抗肿瘤治疗的一个有意义与希望的研究领域。
Therefore a meaningful and hopeful field of anti-tumor treatment study is applying medicine to start the tumor cell apoptosis process.
这些药物的作用机制是阻断酪氨酸激酶启动信号级联反应，后者可以促进肿瘤细胞生长。
The drugs work by preventing an enzyme called tyrosine kinase from launching a signaling cascade that fuels tumor cell growth.
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