采用细胞毒、卤虫致死和微藻克生活性筛选模型，对分离得到的单体化合物进行了初步的生物活性评价。
The primary biological activities of the compounds were evaluated on the models of cell cytoclasis , brine shrimp and growth inhibition of red tide microalgae.
游离二氧化硅致肺组织纤维化机制十分复杂，涉及多种细胞及生物活性物质参与的炎症反应、免疫反应、异物反应、细胞毒作用等。
The mechanism of fibrosis caused by free silica particles involves a lot of cells and reactions including inflammatory reaction, immunological reaction, foreign body reaction and cell toxic action.
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