细小病毒的复制紧紧依赖于和增殖及分化有关的细胞因子，尤其是和细胞周期S-期有关的因子。
The replication of parvovirus is strongly dependent on cellular factors associated with proliferation and differentiation, especially those related to the S-phase of the cell cycle.
结论HCMV的感染改变了细胞内P 53的水平，可能影响其在细胞周期中的正常功能，营造有利于病毒复制的环境。
Conclusion HCMV induces elevated levels of P53, which may affect its normal function in regulating checkpoints of cell cycle, and facilitate its own replication in host cell.
研究表明灵菌红素抑制细胞的增殖，与抑制S期细胞的DNA复制及调控细胞的增殖周期相关。 同时，细胞凋亡的产生与实验药物呈正相关。
The inhibitive effect of prodigiosin on multiplication of cancer cells was related to the inhibiting of DNA replication in S stage and regulating of cell cycle.
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