在这些细胞里，它们被一种尚未被人类完全认识的机制保护着从而免受药物作用。
In these cells it is protected from the drugs by mechanisms that are, as yet, not fully understood.
接下来的几周里这些细胞转移到大脑，由基因遗传机制和相邻细胞的共同作用到达特定的地址。
Over the next several weeks these cells migrated to the brain, to specific destinations determined by genetic cues and interactions with neighboring neurons.
范可尼贫血(FA)病人是由于DNA修复机制——一种有助于我们抵抗癌细胞增长的正常机制——不再充分发挥作用而引起的。
09052109 in patients with Fanconi anaemia (FA), one of the DNA repair mechanisms that normally protects us against cancer no longer works adequately.
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