表明三七皂甙对能量代谢障碍引起的神经细胞损伤有保护作用，这一作用可能与其抗脂质过氧化损伤有关。
It is revealed that PNS can prevent the damage or nerve cells due to energy metabolic disturbance, which may relate to its function of fighting against lipid peroxidation.
结论：朊蛋白106 - 126肽段对分化PC 12细胞具有毒性作用，能量代谢障碍可能是其毒性作用机制之一。
Conclusion: Prion protein 106-126 peptide has cellular toxicity effect on differentiated PC12 cells, and the energy metabolize disorder may be one of the reasons for the toxic mechanism.
结论：老年2型糖尿病患者血粘度增高和红细胞变形能力降低可以增加代谢物质血流传输障碍，加重胰岛素抵抗。
Conclusion: High blood viscosity and impaired erythrocyte deformability in erythrocyte membrane may increase resistance to insulin in aged type 2 diabetes.
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