心脏骤停后使用亚硝酸盐治疗能够减少活性氧生成、改善心脏和神经功能并且通过对线粒体复合物i的可逆性阻滞提高生存率。
Nitrite Therapy After Cardiac Arrest Reduces Reactive Oxygen Species Generation, Improves Cardiac and Neurological Function, and Enhances Survival via Reversible Inhibition of Mitochondrial Complex I.
线粒体功能不良的测定，是通过计算当他们转录自身dna密码到生成必须物质，如蛋白，发生错误的数量。
Mitochondrial dysfunction is measured by counting the number of mistakes they make when they transcribe their DNA code to make essential materials like proteins.
内皮细胞功能紊乱的根本机制在于内皮细胞线粒体活性氧簇(ROS)生成过多。
An underlying mechanism for endothelial dysfunction is the enhanced generation of endothelial mitochondrial reactive oxygen species (ROS).
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