但是，对轻中度收缩性HF患者，螺内酯联合血管紧张素ii受体拮抗剂对左室(LV)逆重构的价值目前还不清楚。
However, the value of combining spironolactone with an angiotensin II receptor blocker on LV reverse remodeling in mild-to-moderate systolic HF is unclear.
依据上述论点，本研究利用AT2受体基因敲出小鼠，观察了AT2受体缺失后是否造成肾素-血管紧张素系统其它成分代偿性紊乱。
In the present study, we investigated whether gene deletion of AT2 receptor causes the compensatory chaos of renin-angiotensin system in mice.
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