细胞间黏附分子- 1 (ICAM - 1)与肾脏缺血再灌注损伤的病理生理密切相关。
It has been well-documented that ICAM-1 implicates in pathophysiology of ischemic-reperfusion injury of the kidney.
涉及的信号通路主要是T细胞受体信号通路、黏附分子、NK介导细胞毒效应等相关通路。
The main signaling pathways involved are the receptor signaling pathway of T cells, the adhesion molecules, and the cytotoxic effect of NK cells.
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