摘要】目的 机械通气时可因气压、容积因素和生物因子释放导 致肺损伤称呼吸机相关性肺损伤(Ventilator-induced lung injury VILI),其病理学基础是广泛的肺泡-毛细血管膜损伤.
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呼吸机相关性肺损伤 ventilator-induced lung injury ; VALI ; ventilator associated lung injury
机械通气相关性肺损伤 VILI ; ventilation related lung injury
输血相关急性肺损伤 transfusion related acute lung injury ; TRALI ; transfusion-related lung injury
通气相关性肺损伤 Ventilator associated Lung Injury ; VALI ; VILI ; ventilator-induced lung injury
气相关性肺损伤 VILI ; ventilator-induced lung injury
通气机相关性肺损伤 venti latorinduced lung injury
而呼吸机相关性肺损伤 ventilator associated lung injury
即呼吸机相关性肺损伤 ventilator associated lung injury
呼吸机相关性肺损伤研究常用大潮气量和高浓度氧策略的动物模型，使用时间相对较短。
Ventilation-induced lung injury is often studied in animal models by using ventilation strategies with high-tidal volumes and high-oxygen concentration over a relatively short period of time.
结论低潮气量通气治疗ARDS动物模型能够减轻炎症诱发细胞因子的释放，减轻机械通气相关性肺损伤。
Conclusion Low tidal volume ventilation can decrease cytokine release and can alleviate ventilator - induced lung injury (VILI) in animal models of ARDS.
目的通过建立过度通气导致呼吸机相关性肺损伤(VILI)大鼠模型，评估不同潮气量对肺损伤的影响。
Objective to establish an experimental model of ventilator-induced lung injury (VILI) in rats through tracheal tube and evaluate the influence of different ventilatory volumes on VILI in rat lung.
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