结论：HCV可以感染滋养层细胞，并导致滋养层细胞的超微结构发生类似黄病毒科病毒感染后的改变。
CONCLUSION: Trophoblastic cells could be infected by HCV, and the cellular ultrastructure changed dramatically following infection of HCV.
绒毛膜滋养层细胞的特性及近期发现的滋养层干细胞，使它们成为一个研究细胞分化和器官形成的理想的遗传学平台。
The feature of HVT together with the recent establishment of trophoblast stem cells makes them an ideal genetic platform to study cell differentiation and organogenesis.
结论：MMP一2、MMP一9参与了正常早孕滋养细胞的侵袭的过程，而TIMP-1、TIMP一2使滋养层细胞以一种可控制的形式侵袭。
Conclusion: MMP-2 and MMP-9 be involved in the invasion of the normal human cytotrophoblast cells, while TIMP-1 and TIMP-2 make this invasion controllable.
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