当动物缺乏PRG - 1基因和溶血磷酯酸受体时，也不会发生癫痫，更多证据说明prg - 1基因是通过磷脂信号起作用的。
Animals lacking both PRG-1 and the LPA receptor didn't have epilepsy either, more evidence that PRG-1 ACTS via the lipid signal.
溶血磷脂酸(LPA)是一种生物活性磷脂介质，主要通过G蛋白偶联受体发挥多种生物学功能。
Lysophosphatidic acid (LPA) is a bioactive phospholipid mediator, which elicits a variety of biological functions mainly, through G-protein coupled receptors.
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