背景与目的哮喘是慢性气道免疫性炎症，以气道淋巴细胞、嗜酸性粒细胞浸润、气道高反应性、粘液产生增加为特征。
Background and Objective Asthma is chronic airway immune inflammation characterized by airway lymphocyte infiltration and eosinophilia, airway hyperresponsiveness (AHR) and mucus hypersecretion.
结论哮喘组大鼠肺组织活性氧产量增加。MT干预可以降低肺组织活性氧产生，降低气道炎症和气道高反应性，这可能是其治疗哮喘的保护机制。
Conclusions MT can decrease airway inflammation and the content of ROS in asthmatic rats, which may be the underlying protective mechanisms of MT against asthma.
气道炎症细胞积聚、气道慢性炎症和气道高反应性是哮喘的三大重要特征。
Accumulation of airway inflammatory cells, chronic airway inflammation and airway hyperresponsiveness are three important features of asthma.
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