结论：去整合蛋白和金属蛋白水解酶10和17的基因表达水平可能不是老龄人易发阿尔茨海默病的生物学基础。

CONCLUSION: The expression levels of ADAM 10 and ADAM 17 in hippocampus of old men may not be a part of biologic foundation of Alzheimer disease.

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为了最大化基因组序列的价值，它们需要用其他类型的生物学数据及彼此进行整合。

To maximize the value of genome sequences they need to be integrated with other types of biological data and with each other.

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微阵列非常适合于整合的系统生物学方式，但是没有一个现有的微阵列数据库是集中于拷贝数变化的。

Microarrays are well suited for the integrative systems biology approach, but none of the existing microarray databases is focusing on copy number changes.

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