方法热休克预处理诱导热休克蛋白产生，然后制作烧伤大鼠急性胃黏膜损伤模型，进行胃黏膜损伤指数评分。

Methods After heat shock proteins (HSPs) were induced by heat shock pretreatment, burn-induced gastric mucosal lesion model was established according to the protocol of Kitajima.

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结论模拟失重可诱导PC 12细胞NO的合成，增加蛋白质氧化损伤程度。

Conclusions Simulated weightlessness may induce the (over) production of NO, and resulted in increased protein oxidative damage in PC12 cells.

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目的探讨内毒素（LPS）诱导的急性肺损伤（ALI）大鼠肺组织甲状腺转录因子-1（TTF-1）、肺表面活性物质相关蛋白-A（SP-A）表达的变化。

Objective To investigate the change of the expression of TTF-1 and SP-A in lung tissue of ALI rats induced by LPS.

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